HPV16 integration probably contributes to cervical oncogenesis through interrupting tumor suppressor genes and inducing chromosome instability

Table 1 The samples collected from HPV positive patients

Sample name	Age (years)	Pathology	Number of validated viral-cellular junctions
S1-2	41	SCC	1
S2-25	45	HSIL	2
C3-64	45	HSIL	1
C4-77	32	HSIL	1
C5-87	49	HSIL	2
S6-95	48	SCC	1
C7-35	44	Normal	0
C8-7	51	HSIL	0
S9-10	38	SCC	0
S10-11	43	SCC	0
C11-60	28	HSIL	0
C12-67	42	SCC	0
C13-75	35	LSIL	0

HSIL: high-grade squamous intraepithelial lesion, LSIL: low grade squamous intraepithelial lesion, SCC: squamous cell carcinoma. The sample ID, age of pateints, Pathology of patients and number of validated viral-cellular junctions in each sample were shown in column 1, 2, 3 and 4 respectively

ISSN: 1756-9966