Fig. 3
From: Role of metabolism in cancer cell radioresistance and radiosensitization methods
A schematic model illustrating the relationship between mitochondrial metabolismand radiation resistance. IR inhibits the oxidative stress of mitochondria, causes abnormal expression of mitochondrial protein and increases the mitochondrial membrane potential, thereby promoting DNA damage repair and inhibiting apoptosis, leading to the occurrence of radioresistance. * was used to represent the targets to enhance radiosensitivity, the corresponding radiosensitizers are indicated in the same color in rectangle