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Fig. 1 | Journal of Experimental & Clinical Cancer Research

Fig. 1

From: LAT2 regulates glutamine-dependent mTOR activation to promote glycolysis and chemoresistance in pancreatic cancer

Fig. 1

LAT2 is upregulated in pancreatic cancer. a The expression level of LAT2 in 71 pancreatic cancer tissue samples was higher than that in the matched paracancerous tissues (*, P < 0.05). b The Wilcoxon signed rank test was applied to compare the difference in LAT2 expression between the cancer and paracancerous tissues. c The expression of LAT2 in pancreatic cancer tissues was evaluated by immunohistochemistry. d The expression of LAT2 in pancreatic paracancerous tissues was evaluated by immunohistochemistry. e Kaplan-Meier survival analysis indicated that the overall survival time of the high-LAT2-level group is shorter than that of the low-LAT2-level group of patients with pancreatic cancer (P = 0.0154)

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