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Fig. 7 | Journal of Experimental & Clinical Cancer Research

Fig. 7

From: Helicobacter pylori CagA promotes epithelial mesenchymal transition in gastric carcinogenesis via triggering oncogenic YAP pathway

Fig. 7

a, b Wound healing assay (a) and Boyden chamber assay (b) were performed in AGS cells infected with H. pylori wild-type strains (PMSS1 or 7.13) and CagA mutants. c AGS cells were co-cultured with CagA+ H. pylori strains PMSS1 or 7.13 in combination with VP treatment, subsequently cells invasion was analyzed by transwell assay. d, e Cell migration were analyzed by wound healing assay (d) and Boyden chamber assay. Data for gene expression are mean ± SEM of 3 independent experiments

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