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Correction: Different p53 genotypes regulating different phosphorylation sites and subcellular location of CDC25C associated with the formation of polyploid giant cancer cells

The Original Article was published on 11 May 2020

Correction: BMC Plant Biol 39, 83 (2020)

https://doi.org/10.1186/s13046-020-01588-w

Following the publication of the original article [1], the authors identified errors in Fig. 3, specifically:

  • Figure 3D - the β-actin of HEY and BT-549 control cells were mistakenly reusede.

  • Figure 3E - the cytoplasm and nuclear protein expression of CDC25C in HEY and BT-549 control and PGCCs with daughter cells the same result was mistakenly placed.

Incorrect Fig. 3

Fig. 3
figure 1

The expression of CHK1, CHK2, PLK1, Aurora A, P53, pCDC25CSer216, and pCDC25CSer198 in HEY and BT-549 control cells and PGCCs with budding daughter cells with and without CDC25C knockdown. a Western blot showed the total protein expression of CHK1, CHK2, PLK1, Aurora A, P53, pCDC25CSer216, and pCDC25CSer198 in HEY and BT-549 control and PGCCs with daughter cells. b The levels of total protein expression of CHK1, CHK2, PLK1, and Aurora A in HEY and BT-549 control and PGCCs with daughter cells, which were transfected with CDC25Ci, siRNA control, and negative control. c Cytoplasmic and nuclear protein expression of CHK1, CHK2, PLK1, Aurora A, P53 in HEY and BT-549 control and PGCCs with daughter cells. d Cytoplasmic and nuclear expression of CHK1, CHK2, PLK1, and Aurora A in HEY and BT-549 control and PGCCs with daughter cells, which were transfected with CDC25Ci, siRNA control, and negative control. e The cytoplasm and nuclear protein expression of pCDC25CSer216 and pCDC25CSer198 in HEY and BT-549 control and PGCCs with daughter cells. Treatment: Cells treated with CoCl2. 1531si: siRNA CDC25C-1531

Correct Fig. 3

Fig. 3
figure 2

The expression of CHK1, CHK2, PLK1, Aurora A, P53, pCDC25CSer216, and pCDC25CSer198 in HEY and BT-549 control cells and PGCCs with budding daughter cells with and without CDC25C knockdown. a Western blot showed the total protein expression of CHK1, CHK2, PLK1, Aurora A, P53, pCDC25CSer216, and pCDC25CSer198 in HEY and BT-549 control and PGCCs with daughter cells. b The levels of total protein expression of CHK1, CHK2, PLK1, and Aurora A in HEY and BT-549 control and PGCCs with daughter cells, which were transfected with CDC25Ci, siRNA control, and negative control. c Cytoplasmic and nuclear protein expression of CHK1, CHK2, PLK1, Aurora A, P53 in HEY and BT-549 control and PGCCs with daughter cells. d Cytoplasmic and nuclear expression of CHK1, CHK2, PLK1, and Aurora A in HEY and BT-549 control and PGCCs with daughter cells, which were transfected with CDC25Ci, siRNA control, and negative control. e The cytoplasm and nuclear protein expression of pCDC25CSer216 and pCDC25CSer198 in HEY and BT-549 control and PGCCs with daughter cells. Treatment: Cells treated with CoCl2. 1531si: siRNA CDC25C-1531

The original article [1] has been corrected.

References

  1. Liu K, Zheng M, Zhao Q, et al. Different p53 genotypes regulating different phosphorylation sites and subcellular location of CDC25C associated with the formation of polyploid giant cancer cells. J Exp Clin Cancer Res. 2020;39:83. https://doi.org/10.1186/s13046-020-01588-w.

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Correspondence to Shiwu Zhang.

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Liu, K., Zheng, M., Zhao, Q. et al. Correction: Different p53 genotypes regulating different phosphorylation sites and subcellular location of CDC25C associated with the formation of polyploid giant cancer cells. J Exp Clin Cancer Res 43, 229 (2024). https://doi.org/10.1186/s13046-024-03155-z

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  • DOI: https://doi.org/10.1186/s13046-024-03155-z