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Fig. 1 | Journal of Experimental & Clinical Cancer Research

Fig. 1

From: SNHG17 promotes colorectal tumorigenesis and metastasis via regulating Trim23-PES1 axis and miR-339-5p-FOSL2-SNHG17 positive feedback loop

Fig. 1

SNHG17 is upregulated in CRC tissues, and its high expression predicted a poor prognosis. a The expression of all SNHGs in CRC tissues of the TCGA database. b The expression of SNHG17 in the pan-cancer TCGA dataset. c The expression of SNHG17 in CRC tissues and NCTs of the TCGA database. d RT-qPCR analysis of SNHG17 expression in CRC tissues from cohort 1. e The percent change in SNHG17 expression in cohort 1 CRC tissues compared with NCTs. f, g Kaplan-Meier survival analyses of the overall survival (f) and disease-free survival (g) in CRC cohort 1 with different expression levels of SNHG17. h The relative expression of SNHG17 in CRC cohort 2. i The rate of SNHG17 upregulation, downregulation and unchanged expression in 107 paired CRC tissues and NCTs. j The relationship between the SNHG17 expression level and clinical pathological parameters of CRC. k, l Kaplan-Meier survival analyses of overall survival (k) and disease-free survival (l) in CRC cohort 2 patients with low or high expression of SNHG17. m Univariate and multivariate regression analyses of independent prognostic factors in CRC patients.

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