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Fig. 1 | Journal of Experimental & Clinical Cancer Research

Fig. 1

From: Hypoxia-induced SKA3 promoted cholangiocarcinoma progression and chemoresistance by enhancing fatty acid synthesis via the regulation of PAR-dependent HIF-1a deubiquitylation

Fig. 1

SKA3 was highly expressed in CCA and associated with poor prognosis. A Differentially expressed genes searched by TCGA, GSE107943 and Tissues. B Prognosis of patients with SKA3 differentially expression in TCGA and GSE107943 datasets. C SKA3 was upregulated in GSE45001. D RT-qPCR analysis of SKA3 expression in CCA tissues. E Western blotting analysis of SKA3 expression in CCA tissues and adjacent normal tissues. F IHC of tissue microarrays showed expression of SKA3 was upregulated in CCA tissues (scale bar: 20μm). G-H Upregulation of SKA3 in CCA was correlated with poor OS and DFS after surgery. *P < 0.05, **P < 0.01, ***P < 0.001

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Journal of Experimental & Clinical Cancer Research

ISSN: 1756-9966

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