Fig. 2From: Hypoxia-induced SKA3 promoted cholangiocarcinoma progression and chemoresistance by enhancing fatty acid synthesis via the regulation of PAR-dependent HIF-1a deubiquitylationSKA3 promoted CCA cell proliferation under hypoxic conditions. A CCK8 assays; B clone formation assays; C Flow cytometry assays; D EdU staining assays indicated that hypoxia-induced SKA3 promoted CCA cell proliferation under hypoxic conditions. *P < 0.05, **P < 0.01, ***P < 0.001Back to article page