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  • Correction
  • Open Access

Correction to: EBV-miR-BART8-3p induces epithelial-mesenchymal transition and promotes metastasis of nasopharyngeal carcinoma cells through activating NF-κB and Erk1/2 pathways

  • 1, 2,
  • 2,
  • 3,
  • 4,
  • 2,
  • 1,
  • 2,
  • 2,
  • 5,
  • 3, 6,
  • 4Email author and
  • 2Email author
Journal of Experimental & Clinical Cancer Research201938:34

https://doi.org/10.1186/s13046-019-1022-5

  • Received: 13 December 2018
  • Accepted: 24 January 2019
  • Published:

The original article was published in Journal of Experimental & Clinical Cancer Research 2018 37:283

Correction to: Journal of Experimental & Clinical Cancer Research (2018) 37:283.

Doi 10.1186/s13046-018-0953-6

Following publication of the original article [1], the authors reported two errors in the article.
  • In the caption of Fig. 1c the sentence “The 20 most highly upregulated EBV BART miRNAs identified between NPC specimens and normal nasopharyngeal mucosal specimens” should instead read “The highly upregulated EBV BART miRNAs identified between NPC specimens and normal nasopharyngeal mucosal specimens”.

  • In Fig. 3a, the first image in the BART8-3p group was inadvertently imported and replaced with the original one in the NC group. A corrected version of Fig. 3 is shown in this Correction.

Fig. 3
Fig. 3

Overexpression of EBV-miR-BART8-3p promotes lung metastasis of NPC in vivo. a Nude mice were intravenously injected with SUNE-1-BART8-3p cells or control vector-transfected SUNE-1 cells via the tail veins, and were sacrificed 6 weeks post-injection. Representative images in vivo were obtained by the whole-body imaging system. b Representative images of metastatic nodules in mouse lungs. c Number of metastatic nodules in mouse lungs. d Weight of mouse lungs. *P < 0.05

Notes

Declarations

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

Authors’ Affiliations

(1)
Fujian Medical University, Fuzhou, 350108, Fujian Province, China
(2)
Department of Radiation Oncology, Fujian Cancer Hospital & Fujian Medical University Cancer Hospital, Fuzhou, 350011, Fujian Province, China
(3)
Laboratory of Immuno-Oncology, Fujian Cancer Hospital & Fujian Medical University Cancer Hospital, Fuzhou, 350011, Fujian Province, China
(4)
Department of Genetics and Genomic Sciences, Icahn Institute of Genomics and Multiscale Biology, Mount Sinai Center for Transformative Disease Modeling, Icahn School of Medicine at Mount Sinai, 1470 Madison Avenue, New York, NY 10029, USA
(5)
State Key Laboratory for Emerging Infectious Diseases, Department of Microbiology and the Collaborative Innovation Center for Diagnosis and Treatment of Infectious Diseases, The University of Hong Kong, Hong Kong, SAR, China
(6)
Fujian Provincial Key Laboratory of Translational Cancer Medicine, Fuzhou, 350014, Fujian, China

Reference

  1. Lin, et al. EBV-miR-BART8-3p induces epithelial-mesenchymal transition and promotes metastasis of nasopharyngeal carcinoma cells through activating NF-κB and Erk1/2 pathways. J Exp Clin Cancer Res. 2018;37(283). https://doi.org/10.1186/s13046-018-0953-6.

Copyright

© The Author(s). 2019

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