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Correction to: ZNF326 promotes malignant phenotype of glioma by up-regulating HDAC7 expression and activating Wnt pathway

  • The original article was published in Journal of Experimental & Clinical Cancer Research 2019 38:40

Correction to: J Exp Clin Cancer Res (2019) 38:40

https://doi.org/10.1186/s13046-019-1031-4

In the original publication of this manuscript [1], the author mislabeled the CTL group and ZNF326 group in Fig.2-I,J (MTT result). The revised Fig. 2 is shown below.

Fig. 2
figure1

Impact of ZNF326 expression on the proliferation and invasiveness of glioma cells in vitro. ZNF326 overexpression significantly enhanced the colony formation (a, b), invasiveness (e, f, magnification-400×), and proliferation (i, j) of U87 and U251 glioma cell lines. Conversely, ZNF326 knockdown significantly inhibited colony formation (c, d), invasiveness (g, h, magnification-400×), and proliferation (k, l) of U87 and U251 glioma cell lines. CTL: control group. Each experiment was performed in triplicate. Columns: mean numbers. Bars: S.D. (*: P < 0.05; **: P < 0.01; ***: P < 0.001)

The authors sincerely apologize for the inconvenience caused to the readers.

Reference

  1. 1.

    Yu X, et al. ZNF326 promotes malignant phenotype of glioma by up-regulating HDAC7 expression and activating Wnt pathway. J Exp Clin Cancer Res. 2019;38:40. https://doi.org/10.1186/s13046-019-1031-4.

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Correspondence to Minghao Wang.

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Yu, X., Wang, M., Wu, J. et al. Correction to: ZNF326 promotes malignant phenotype of glioma by up-regulating HDAC7 expression and activating Wnt pathway. J Exp Clin Cancer Res 39, 14 (2020). https://doi.org/10.1186/s13046-020-1526-z

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