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Correction: AQP5 complements LGR5 to determine the fates of gastric cancer stem cells through regulating ULK1 ubiquitination
Journal of Experimental & Clinical Cancer Research volume 41, Article number: 342 (2022)
Correction: J ExpClin Cancer Res 41, 322 (2022)
Following the publication of the original article , one of the readers spotted an error in the supplementary materials. The Supplementary figures of Additional file 1 is missing and the e-files for Additional file 1 and Additional file 2 were interchanged.
The correction does not have any effect on the results or conclusions of the paper. The original article has been corrected.
Zhao R, He B, Bie Q, et al. AQP5 complements LGR5 to determine the fates of gastric cancer stem cells through regulating ULK1 ubiquitination. J Exp Clin Cancer Res. 2022;41:322. https://doi.org/10.1186/s13046-022-02532-w.
Additional file1: Figure S1.
Expression of marker genes in gastric cancer tissue epithelial/stem cells and cultured adherent/spheroid cells. Figure S2. AQP5 expression in AGS/HGC-27/GES-1 spheroids and adherent cells. Figure S3. AQP5 promotes gastric cancer development in vitro and in vivo. Figure S4. Expression of AQP5 in GC-CSCs. Figure S5. AQP5 promotes the stemness of GC-CSCs. Figure S6. Effect of AQP5 on LGR5 expression. Figure S7. Cellular pathways affected by AQP5. Figure S8. ATG7 is the key regulator of GC cell autophagy. Figure S9. AQP5 affects key autophagy proteins. Figure S10. AQP5 promotes malignant behaviors of GC-CSCs by regulating K63-mediated ubiquitination of ULK1. Figure S11. Interaction of AQP5, TRIM21 and ULK1. Figure S12. AQP5 promotes self-renewal via TRIM21 in GC-CSCs.
Additional file 2: Supplementary Table 1.
Sequences of real-time PCR primers. Supplementary Table 2. Primary Antibodies. Supplementary Table 3. Second Antibodies. Supplementary Table 4. ShRNA or siRNA Oligonucleotides. Supplementary Table 5. Tumorigenicity of knockdown AQP5 and control group. Supplementary Table 6. Tumorigenicity of knockdown AQP5 and control group. Supplementary Table 7. Tumorigenicity of knockdown AQP5 and LGR5. Supplementary Table 8. Differentially expressed genes between exogenous overexpression of AQP5 and control group. Supplementary Table 9. Differentially expressed genes between exogenous overexpression of AQP5 and control group. Supplementary Table 10. Identification of the AQP5 protein complex by mass spectrometry.
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Zhao, R., He, B., Bie, Q. et al. Correction: AQP5 complements LGR5 to determine the fates of gastric cancer stem cells through regulating ULK1 ubiquitination. J Exp Clin Cancer Res 41, 342 (2022). https://doi.org/10.1186/s13046-022-02557-1