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Correction: 3-O-acetyl-11-keto-β-boswellic acid exerts anti-tumor effects in glioblastoma by arresting cell cycle at G2/M phase
Journal of Experimental & Clinical Cancer Research volume 41, Article number: 236 (2022)
Correction: J Exp Clin Cancer Res 37, 132 (2018)
https://doi.org/10.1186/s13046-018-0805-4
Following publication of the original article [1], an error was identified in Fig. 3; specifically:
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Figure 3e and 3f, panels “U251-MG 48h Control” and “U87-MG 48h Control” were inadvertently duplicated
AKBA induces mitochondria-dependent apoptosis in U251 and U87-MG cells in a dose-dependent manner. Flow cytometry using Annexin V-FITC staining shows that AKBA increases apoptosis of U251 (a) and U87-MG (b) cells. Flow cytometry using JC-1 staining shows that AKBA reduces mitochondrial membrane potential in U251(c) and U87-MG (d) cells. Flow cytometry using caspase 3/7 live-cell staining shows that AKBA increases the activity of caspase 3/7 in U251 (e) and U87-MG (f) cells. Western blotting results show that AKBA induces expression of cleaved-caspase 3 and cleaved-PARP in U251 (g) and U87-MG (h) cells. The experiments were performed in triplicate
The correction does not have any effect on the results or conclusions of the paper.
Reference
Li W, Liu J, Fu W, et al. 3-O-acetyl-11-keto-β-boswellic acid exerts anti-tumor effects in glioblastoma by arresting cell cycle at G2/M phase. J Exp Clin Cancer Res. 2018;37:132. https://doi.org/10.1186/s13046-018-0805-4.
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Li, W., Liu, J., Fu, W. et al. Correction: 3-O-acetyl-11-keto-β-boswellic acid exerts anti-tumor effects in glioblastoma by arresting cell cycle at G2/M phase. J Exp Clin Cancer Res 41, 236 (2022). https://doi.org/10.1186/s13046-022-02454-7
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DOI: https://doi.org/10.1186/s13046-022-02454-7