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Correction: 3-O-acetyl-11-keto-β-boswellic acid exerts anti-tumor effects in glioblastoma by arresting cell cycle at G2/M phase

The Original Article was published on 03 July 2018

Correction: J Exp Clin Cancer Res 37, 132 (2018)

https://doi.org/10.1186/s13046-018-0805-4

Following publication of the original article [1], an error was identified in Fig. 3; specifically:

  • Figure 3e and 3f, panels “U251-MG 48h Control” and “U87-MG 48h Control” were inadvertently duplicated

Fig. 3
figure 1

AKBA induces mitochondria-dependent apoptosis in U251 and U87-MG cells in a dose-dependent manner. Flow cytometry using Annexin V-FITC staining shows that AKBA increases apoptosis of U251 (a) and U87-MG (b) cells. Flow cytometry using JC-1 staining shows that AKBA reduces mitochondrial membrane potential in U251(c) and U87-MG (d) cells. Flow cytometry using caspase 3/7 live-cell staining shows that AKBA increases the activity of caspase 3/7 in U251 (e) and U87-MG (f) cells. Western blotting results show that AKBA induces expression of cleaved-caspase 3 and cleaved-PARP in U251 (g) and U87-MG (h) cells. The experiments were performed in triplicate

The correction does not have any effect on the results or conclusions of the paper.

Reference

  1. Li W, Liu J, Fu W, et al. 3-O-acetyl-11-keto-β-boswellic acid exerts anti-tumor effects in glioblastoma by arresting cell cycle at G2/M phase. J Exp Clin Cancer Res. 2018;37:132. https://doi.org/10.1186/s13046-018-0805-4.

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Correspondence to Jinhua Wang, Tengfei Ji or Guanhua Du.

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Li, W., Liu, J., Fu, W. et al. Correction: 3-O-acetyl-11-keto-β-boswellic acid exerts anti-tumor effects in glioblastoma by arresting cell cycle at G2/M phase. J Exp Clin Cancer Res 41, 236 (2022). https://doi.org/10.1186/s13046-022-02454-7

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  • DOI: https://doi.org/10.1186/s13046-022-02454-7